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TABLE 2 Effect of phytic acid on serum iron and TIBC among different experimental groups

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This paradox arises from the inherent complexity of MS pathogenesis which we argue is driven by a “convolution” of relapsing/acute adaptive autoimmunity and chronic cytodegeneration (cyto-is used here rather than neuro-to underscore the point that myelin/oligodendrocytes are the likely primary targets of this process), including an elaborate innate immune response (Figure 1)